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Semaglutide and liraglutide both showed psychiatric and gut signals in Korea

Thirteen hospitals, ten outcomes, several elevated. The widest intervals sit under the biggest numbers, and the control group never started treatment.

Glenn Torres6 min read
Semaglutide vs people not starting it1.0gut dysmotilitydepressionanxietyvisionRead the bars, not the dots. Two run past the edge.

Ten safety outcomes were tested across 13 hospitals, and several came back elevated [1]. Before any of the numbers, the comparator deserves attention, because it decides what the numbers can mean. Everyone in the control group was a matched person who did not start a GLP-1 — and the reasons a person starts a weight drug are not the reasons another person does not — the comparator problem this site set out in how the comparator decides the depression answer.

For semaglutide, psychiatric disorders overall came in at a hazard ratio of 2.02 (95% CI 1.30 to 3.14), anxiety at 2.39 (1.22 to 4.71), depressive disorder at 3.42 (1.51 to 7.74), gastrointestinal dysmotility or obstruction at 3.91 (1.42 to 10.82) and vision impairment at 1.58 (1.04 to 2.41). For liraglutide, with a larger sample, psychiatric disorders were 1.66 (1.36 to 2.03), hepatic impairment 1.46 (1.16 to 1.83), pancreatitis, cholangitis or cholelithiasis 1.40 (1.18 to 1.68) and vision impairment 1.34 (1.16 to 1.55).

The comparator problem is worth stating precisely rather than waving at. Someone who starts a weight-management drug has usually just seen a doctor, is being monitored, and is under a degree of distress about their weight. Their matched non-initiator is not necessarily doing any of those things. Higher recorded rates of anxiety and depression in a group with more clinical contact can reflect more diagnosis, not more disease. Nothing in a matched design removes that.

This is exactly the contrast with the cohort this site covered in the study that tested its own bias, which used an active comparator — people taking a different drug — precisely so that both groups were under care. That choice does not make this study wrong. It makes the two studies answer different questions.

The authors add their own caveat: semaglutide-associated vision impairment and liraglutide-associated hepatic impairment lost significance in some sensitivity analyses. The vision signal in particular joins a literature this site has followed in the sudden vision loss reports, where the same pattern recurs: a real signal, repeatedly detected, never yet cleanly sized.

Frequently asked

Does this show GLP-1 drugs cause depression?
No. It shows higher recorded rates against matched people who did not start one, with an interval from 1.51 to 7.74. Groups with more clinical contact also receive more diagnoses, which a matched design cannot separate out.
Which signals were strongest?
For semaglutide, gastrointestinal dysmotility or obstruction (HR 3.91) and depressive disorder (3.42) — both with very wide intervals. For liraglutide, psychiatric disorders (1.66) and pancreatitis-related outcomes (1.40), with tighter ones.
Did any findings not hold up?
Yes. The authors report that semaglutide-associated vision impairment and liraglutide-associated hepatic impairment were not significant in some sensitivity analyses.

Sources

  1. [1] Park J, Kim JH, Kim YS, Lim G, Song HJ, Rhee SY (2026). GLP-1 Receptor Agonists for Weight Loss and Risk of Major Safety Outcomes: A Multicentre Cohort Study Diabetes, Obesity and Metabolism. PMID 42410329

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