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Fewer clots on tirzepatide, and a study that tested its own weakness

The effect is small and the method is unusually careful. One internal detail complicates it: the lung clots moved and the leg clots did not.

Ruth Alvarez7 min read
Tirzepatide vs other GLP-1 drugs, 1 yearblood clots overall0.90pulmonary embolism0.88deep vein thrombosis0.96death, any cause0.90Clots in the lung moved. Clots in the leg did not.

Most claims studies report an association and leave the reader to guess how much unmeasured confounding would erase it. This one tests that directly, which makes it worth reading for its method as much as its result [1]. Other comparisons between the same two drugs are in the semaglutide and tirzepatide comparison.

Two instruments were used. A negative control outcome is something the drug could not plausibly affect: if the analysis finds an effect there, the method is manufacturing associations. An E-value asks how strong an unmeasured confounder would have to be, in association with both treatment and outcome, to explain away the result entirely. Together they convert “this might be confounded” from an objection into a quantity.

The finding itself is modest. Venous thromboembolism over one year gave a hazard ratio of 0.90 with a confidence interval of 0.83 to 0.98. An upper bound that close to 1 across 701,374 people describes a small effect measured precisely — the precision comes from the sample size, not from the size of the difference.

All-cause mortality also fell, at 0.90 — the same magnitude as the primary outcome. In a one-year study of blood clots, a mortality difference of equal size suggests the two arms differ in general health rather than specifically in clotting risk, which is the pattern described at greater length in six unrelated complications all halved. That the authors ran negative controls makes this less likely than it otherwise would be, without eliminating it.

For a reader the practical content is small and the methodological content is not. A 10% relative reduction in clots between two drugs in the same class is not a reason to choose between them, and it sits alongside the other small tirzepatide advantages this site has covered in the glaucoma comparison and the carpal tunnel cohort. What is genuinely useful is that a large claims study can quantify its own fragility, and that most do not.

Frequently asked

Is tirzepatide safer than other GLP-1 drugs for blood clots?
The hazard ratio was 0.90 with an upper bound of 0.98 — a small difference measured precisely in 701,374 people. It is not large enough to drive a choice between the two.
What is an E-value?
A measure of how strong an unmeasured confounder would have to be, in its association with both the treatment and the outcome, to fully explain away a reported result.
Why does the deep vein thrombosis result matter?
Most pulmonary emboli begin as deep vein thromboses. A treatment that reduces lung clots without reducing leg clots is mechanistically difficult to explain, which is a reason for caution.

Sources

  1. [1] Wu JY, Lee KW, Huang SC, Chang HY, Lin YM (2026). Comparative effectiveness of tirzepatide versus GLP-1 receptor agonists on the risk of venous thromboembolism in patients with obesity: a real-world cohort study Frontiers in Medicine. PMID 42239959

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